Download Clinical Arrhythmology and Electrophysiology: A Companion to by Ziad Issa MD, John M. Miller MD PDF
By Ziad Issa MD, John M. Miller MD
With its distinctive, singular specialize in the scientific point of cardiac arrhythmias, Clinical Arrhythmology and Electrophysiology: A spouse to Braunwald's middle Disease makes it effortless to use modern most modern directions for prognosis and therapy. a professional writer crew presents clear, clinically centred guidance on all kinds of cardiac arrhythmias, together with practical ideas for coping with complicated patients.
- Find the data you would like quickly
- Access the totally searchable contents online
- Fully comprehend the reason for remedy of particular arrhythmias
with a constant association in all chapters, written to a template that indicates each arrhythmia variety in the same manner.
at www.expertconsult.com, as well as downloadable photographs and dynamic movies.
with sensible concepts which are grounded within the latest easy technology.
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- View movies of 27 key ideas online
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with new chapters on molecular mechanisms of cardiac electric job, cardiac ion channels, ventricular tachycardia in nonischemic dilated cardiomyopathy, epicardial ventricular tachycardia, ventricular arrhythmias in hypertrophic cardiomyopathy, ventricular arrhythmias in inherited channelopathies, ventricular arrhythmias in congenital center illness, atrial arrhythmias in congenital center illness, and issues of catheter ablation of cardiac arrhythmias.
, together with optical mapping of reentrant ventricular arrhythmias, third-dimensional mapping of arrhythmias utilizing varied mapping and navigation modalities, and fluoroscopy photographs illustrating concepts for electrophysiologic catheter positioning, atrial septal puncture, and pericardial entry.
similar to mechanisms of arrhythmias, electrophysiologic checking out, mapping and navigation modalities, ablation strength assets, sinus node disorder, conduction disturbances, atrial tachyarrhythmias, preexcitation syndromes and all kinds of ventricular and supraventricular tachycardias.
Tackle the scientific administration of cardiac arrhythmias with self belief with the main up to date assistance from the specialists you trust.
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Additional info for Clinical Arrhythmology and Electrophysiology: A Companion to Braunwald's Heart Disease
3 Short QT Syndrome A gain-of-function mutation of KCNJ2 has been identified and linked to SQTS type 3 (SQT3). 37 Familial Atrial Fibrillation A gain-of-function KCNJ2 mutation has been linked to familial AF. The affected members had normal QT intervals. 3,34 ACQUIRED DISEASES IK1 is downregulated in patients with severe heart failure and cardiomyopathy. The ventricular myocytes from patients with idiopathic dilated cardiomyopathy exhibit decreased channel activity, longer action potential duration, and a lower resting Em than those from patients with ischemic cardiomyopathy.
Although organic T-type Ca2+ channel blockers (mibefradil) result in marked decrease in firing frequency of sinus node cells in clinical studies, the possibility that these blockers affect other ionic currents, including ICaL, cannot be entirely excluded. In fact, it is has not yet been determined whether ICaT exists functionally in atrial, ventricular, and sinoatrial node cells in the human heart. Further studies are necessary to clarify CARDIAC ION CHANNELS α1C subunit. The combined use of Ca2+ channel blockers can enhance or weaken the block effect because, at least in part, of the different binding sites for those drugs.
6, calmodulin). On the luminal (sarcoplasmic reticulum) side, RyR2 forms a part of a large quaternary complex with calsequestrin (CASQ2), triadin, and junctin. 57-60 Cardiac RyR2 functions as a ligand-activated ion channel that activates (opens) on Ca2+ binding. However, the exact structural determinants of RyR gating are as yet unknown. RyR2 is normally closed at low cytosolic diastolic Ca2+ concentrations (approximately 100 to 200 nM). 50,59,60 CH The precise juxtaposition of the sarcolemmal specialized invagi- 2 nations (known as T tubules) and sarcoplasmic reticulum forms specific junctional microdomains, creating a 10- to 12-nM gap, known as the dyadic cleft.